The Remnant Online

Health => Healthful Living => Topic started by: Richard Myers on February 16, 2004, 03:57:00 PM

Title: Missing Link to Alzheimer's?
Post by: Richard Myers on February 16, 2004, 03:57:00 PM
Italian scientists have discovered what appears to be a new form of Mad Cow disease in cattle. We have been concerned for some time that CJD that is said to be sporadic and not caused by eating diseased animals is in fact transmitted from diseased animals.

Both the human and cattle diseases cause holes to form in the brain. The Italian researchers found that, in addition to the holes, two cows had an accumulation of amyloid plaque in their brains. Amyloid plaques are an indication of Alzheimer's Disease in humans. They have also been found in people with sporadic CJD but had not been found in cattle.

This is the first scientific indication that I have come across that reveals a possible connection between Alzheimer's Disease and infections in animals. This information ought to cause great concern to those who are still eating animal products.

Title: Missing Link to Alzheimer's?
Post by: JimB on February 17, 2004, 04:48:00 AM
If you don't have to eat it but simply be exposed to animals, it would interesting to see a study done to see if farmers and/or veternarians have a higher rate of these kinds of diseases.
Title: Missing Link to Alzheimer's?
Post by: Richard Myers on February 17, 2004, 05:19:00 PM
Brother Jim, the infectious agent, as I understand does not have to be ingested. My guess is that the amount of infective agent is increased considerably when it is eaten versus a vet who may or may not come in direct contact with infectious material. I don't think there is any question about the infective nature of the prions and that it can infect in many ways that would allow it access to the blood stream.
Title: Missing Link to Alzheimer's?
Post by: Richard Myers on January 30, 2005, 08:24:00 PM
Many have wondered how their loved ones developed Ahlzeimers. Like sporadic CJD it just happens or it is the result of aluminum. No, neither of these diseases just happen an no, aluminum cannot be shown to cause it. I have suspected for a long time that both diseases come from eating the flesh or products of animals with spongiform disease.

We now have found "mad cows" with lesions similar to the ones found in Ahlzeimer patients. I believe this is the evidence we needed to begin to discuss a relationship between the two. Sporadic CJD and Ahlzeimers are caused, they don't just happen. I believe we shall soon find out that they are both caused by infections from animals with a spongiform disease.

I have been looking for the evidence of such a connection for some time. Today, I believe I have found it. There are those who have been aware of this for over thirty years. It is sad that the public has not been informed.

Almost thirty years ago the Agricultural Research Council (ARC) at its meeting on October 12, 1976 stated "The USDA concluded that it could 'no longer justify or permit scrapie-blood line and scrapie-exposed sheep and goats to be processed for human or animal food at slaughter or renderng plants.' (ARC 84/77).The problem is emphasized by the finding that some strains of scrapie produce lesions identical to the ones which characterise the human dementias."

It is very interesting to note that we see here something that ought to interest us all. That the lesions in human dementia (Ahlzeimers) correspond to the lesions produced by scrapie is the "smoking gun" that I believe will be found to explain a cause of Ahlzeimers.

Those who have concern and love for humanity need to take this information to heart. There is no safety in eating the flesh of animals nor in eating any animal product. The government is not to be trusted when it comes to our health. They have other concerns that compete with human and animal health.

Title: Missing Link to Alzheimer's?
Post by: Richard Myers on April 20, 2006, 09:22:00 PM
For many years I have suspected that Scrapie and BSE along with other TSEs are the cause of Alzheimer's. When the study found the amyloid plaques in the mad cows that was enough for me to say the evidence is rather significant.

Now, we have a human that manifests the amyloid plaques and he was diagnosed with CJD, the human form of mad cow disease. Yes, many would like to point to another "unknown" cause of the plaques, but the evidence is very suggestive that we have another link to the cause of Alzheimer's.

"We report the case of a 28 year old man who had received a cadaverous dura mater graft after a traumatic open skull fracture with tearing of the dura at the age of 5 years. A clinical suspicion of Creutzfeldt-Jakob disease (CJD) was confirmed by a brain biopsy 5 months prior to death and by autopsy, thus warranting the diagnosis of iatrogenic CJD (iCJD) according to WHO criteria. Immunohistochemistry showed widespread cortical depositions of disease associated prion protein (PrPsc) in a synaptic pattern, and western blot analysis identified PrPsc of type 2A according to Parchi et al. Surprisingly, we found Alzheimer-type senile plaques and cerebral amyloid angiopathy in widespread areas of the brain. Plaque-type and vascular amyloid was immunohistochemically identified as deposits of beta-A4 peptide. CERAD criteria for diagnosis of definite Alzheimer’s disease (AD) were met in the absence of neurofibrillar tangles or alpha-synuclein immunoreactive inclusions. There was no family history of AD, CJD, or any other neurological disease, and genetic analysis showed no disease specific mutations of the prion protein, presenilin 1 and 2, or amyloid precursor protein genes. This case represents (a) the iCJD case with the longest incubation time after dural grafting reported so far, (b) the youngest documented patient with concomitant CJD and Alzheimer-type neuropathology to date, (c) the first description of Alzheimer-type changes in iCJD, and (d) the second case of iCJD in Austria. Despite the young patient age, the Alzheimer-type changes may be an incidental finding, possibly related to the childhood trauma." Journal of Neurology, Neurosurgery, and Psychiatry 2006;77:413-416;doi:10.1136/jnnp.2005.070805
BMJ

[This message has been edited by Richard Myers (edited 04-20-2006).]

Title: Missing Link to Alzheimer's?
Post by: Michelle on April 20, 2006, 10:20:00 PM
So what do we say about vegetarians who get Alzheimers?  Is there a study following this line of reasoning?  Is there an anecdotal evidence of vegans not being susceptible?  Or of vegans who have developed Alzheimers?

M

Title: Missing Link to Alzheimer's?
Post by: Liane H on April 21, 2006, 04:04:00 AM
Hi Michelle:

According to what I have read this morning the link between vegetarians an Alzeimers disease seems to be grounded around low B12.

The statistics states though there is a far more link between Alzeimer's with meat than with vegetarians. Also the link with cholesteral, diabetes and blood pressure problems with meat was even higher of Alzeimer's disease found.

There was also a study that said there was a higher risk of Alzeimer's with those that consumed a high amount of soy products.

No question about it, there is much confusion and no sure answer regarding any of this and the Alzeimer's Association says there have been no conclusive study regarding what really causes Alzeimer in people. They even state there is a genetic factor link to this disease as well.

Liane, the Zoo Mama


Title: Missing Link to Alzheimer's?
Post by: Richard Myers on April 21, 2006, 10:46:00 AM
Sister Michelle, that is a good question. It is one that we need to consider when trying to pinpoint the causes of such dreaded diseases.

In the case of CJD, the human form of BSE (mad cow)we know that the route of transmission is more than just diet. So, the same would hold true with Alzheimer's. If it is indeed infectious, then it is not just the diet that can infect the person.

It is my understanding that the prions are highly infectious. One of my concerns is that they have gotten into many products that people are not aware of. There are strict vegetarians that have eaten candy made with gelatin. And there are strict vegetarians that have taken vitamins and other supplements in capsuls made with gelatin. Gelatin is a very high risk matertial that was allowed into the U.S. from England when the BSE problem was at its  height.

Since the manufacturing of the products that may contain this infectious prion never reaches temperatures that can inactivate the infectious nature of the material, we can be exposed to many products that may contain them.

A route of transmission that concerns me that I feel is a high risk, especially to children is pet food. We have reached the point in this world's history where we must move beyond not eating infected animal products, but take greater care in coming into contact with them. Cleanliness has never been more important.

Another route of transmission of disease that is becoming more of a problem is our water supply. BSE and other TSEs including Chronic Wasting Disease is a threat to our water supply. Much drinking water comes from surface sources such as rivers and streams. They are all contaminated to various degrees from many infectious materials. But, TSEs is not a subject that I have heard discussed by those responsible for water treatment. It appears to me to be a very serious health threat.

Chronic wasting disease (mad cow disease in deer and elk) is spreading rapidly throughout the U.S. It must be in about half the states now and moving quickly into those not infected. Cattle are infected with BSE and they pose a problem for our water supply, but I see deer and elk as a great risk also. One of the suspected routes of transmission amongst the deer and elk is thought to be urine. While the risk appears to be very low from water supplies, I don't think we can ignore the risk.

As an example, ecoli is another very infectious disease that comes from cattle. Does it pose a risk to our water supply? You bet it does. If you live in Canada or the U.S. and there are cattle in the neighborhood. If your source of water is a well, then you need to get it tested regularly. It is a very deadly disease for humans, especially the elderly and the young.

Ecoli is easily treated in the water, but not so prions that are infectious. I takes higher amounts of chemicals to inactivate the infectious nature and these levels are too high for the safe drinking water. It is a bridge that soon we shall have to deal with as the epidemic of TSEs continues to grow.

If we believe that Jesus is soon to return, and I do, then we can expect to see much pestilence. When we love the Lord and give our hearts to Him, He gives us wisdom and power. The wisdom is the light He has given in these matters regarding the infectious nature of animal products. The power is given to walk in that light in spite of our depraved appetites.

We ought not to fear when we have done all that we can do. We have this promise: "I will say of the LORD, He is my refuge and my fortress: my God; in him will I trust. Surely he shall deliver thee from the snare of the fowler, and from the noisome pestilence.  He shall cover thee with his feathers, and under his wings shalt thou trust: his truth shall be thy shield and buckler.  Thou shalt not be afraid for the terror by night; nor for the arrow that flieth by day;  Nor for the pestilence that walketh in darkness; nor for the destruction that wasteth at noonday. A thousand shall fall at thy side, and ten thousand at thy right hand; but it shall not come nigh thee."

Title: Missing Link to Alzheimer's?
Post by: Curt on May 06, 2006, 01:58:00 PM

Alzheimer's Theory Set for Drug Test (Diabetes Link)
May 3, 2006

WASHINGTON (AP) -- A provocative new theory suggests that one root cause of Alzheimer's disease is linked to diabetes -- a theory about to be tested in thousands of Alzheimer's patients given the diabetes drug Avandia in hopes of slowing brain decay.

It's a scary scenario: Alzheimer's already is expected to skyrocket as the population grays, rising from 4.5 million sufferers today to a staggering 14 million by 2050. If the new theory is right, the nation's current obesity-fueled epidemic of Type 2 diabetes could worsen that toll.

But proponents see potential good news: If diabetic-like changes in the way brain cells use sugar to generate energy truly trigger Alzheimer's in at least some patients, then maybe doctors could intervene early and slow down that degeneration.

A preliminary experiment involving 511 Alzheimer's patients found signals that Avandia might help -- albeit in people who lack a gene that spurs more aggressive Alzheimer's.

Those results, combined with other evidence that the diabetes pathway is important, have Avandia maker GlaxoSmithKline poised to open three Phase III clinical trials this summer to test whether the diabetes drug, also called rosiglitazone, might protect certain patients' brains.

Diabetes has long been listed a risk factor for Alzheimer's later in life because it damages blood vessels that supply the brain.

The Avandia research suggests a more insidious connection: that Alzheimer's can be silently triggered when brain cells can't properly use their main fuel, sugar -- just as Type 2 diabetes is triggered when insulin gradually loses its ability to process sugar body-wide.

"When they're in an insulin-resistant state, it does not just affect the body, it affects the brain as well," explains Suzanne Craft of the Veterans Affairs Puget Sound Health Care System, who led the initial research.

There are 18 million Type 2 diabetics, considered to have two to five times a non-diabetic's risk of developing Alzheimer's -- if they live long enough, into the 60s and 70s when Alzheimer's typically strikes, Craft says. Type 2 diabetes often leads to heart disease or other ailments that kill before then.

Avandia, and the competing drug Actos, treat Type 2 diabetes by resensitizing the body to insulin.

Don't use Avandia for Alzheimer's until that question is settled, cautions Glaxo's Allen Roses, a highly regarded Alzheimer's researcher who, before joining the pharmaceutical company, discovered the brain disease's main genetic link.

He published the diabetes hypothesis in the medical journal Alzheimer's & Dementia last week.

If it pans out, "one thing we can do is, possibly, slow down the onset of the disease showing up," Roses says. But for now, "it's a hopeful experiment that's in progress."

Yet it's generating intense interest.

"This is an exciting new approach," Yadong Huang of the University of California, San Francisco, wrote in an accompanying review of Roses' hypothesis, which is backed by genetics research from Huang's own lab.

"I don't think this is hype for rosiglitazone," adds Dr. Sam Gandy, director of the Farber Institute for Neurosciences at Philadelphia's Thomas Jefferson University and an Alzheimer's Association spokesman. "This does dovetail with some existing knowledge."

No one knows what causes Alzheimer's creeping brain degeneration. Today's drugs only temporarily help symptoms. Now, researchers are hunting new ones that target the disease's hallmark, a sticky gunk called beta-amyloid that clogs up, and probably kills, neurons.

The new theory: The metabolism of neurons' internal power factors, called mitochondria, go awry so that those cells don't use enough sugar. That eventually leads to impaired brain cell function, including the buildup of that gunky beta-amyloid. It also means that neurons in youth and middle age don't sprout enough communication connections, providing less "cognitive reserve" once their neurons start dying off.

Among the evidence Roses cites:

   * Decades before dementia symptoms begin, PET scans of people who carry an Alzheimer's-linked gene show their brain cells underuse sugar.
   * Mice engineered to develop Alzheimer's-like disease experience the metabolism changes before beta-amyloid plaques appear -- and insulin-sensitizing drugs enhance their brains' sugar use.
   * Researchers who tracked 140,000 diabetic veterans found those who received insulin-resensitizing drugs were less likely to be diagnosed with Alzheimer's years later.

Then there's that preliminary Avandia study. Alzheimer's patients showed modest improvement in cognitive function -- unless they carried the Alzheimer's-linked gene called ApoE4 that spurs more aggressive disease. The new studies will try to prove if that benefit was real.

Copyright 2006 The Associated Press. All rights reserved.
   .    
InteliHealth

Title: Missing Link to Alzheimer's?
Post by: stephen on May 10, 2006, 09:46:00 PM
It is thought that the same spongiform bacteria is responsible for the increase in Lou Gherig's Disease also.  That spongiform bacteria is called Mad Cow in cattle.  It is called Chronic Wasting Disease in Deer.  It is also found in swine, sheep, goats.  It goes by a different name in each species.  There have been deaths caused by this bacteria in humans but under a different name.  A dentist told me of one death last year here in Michigan.  Two physician friends told me of two cases last summer in northern Michigan.  I don't remember the name it was diagnosed by.  But one thing for sure, the US Beef Industry is a powerful lobby and the people are not going to be informed too much.  At the same time people are dying and more is known about the links between this disease and the likes of alzheimers and LGD, etc., our government is very interested in opening foreign markets more to US Beef.  Don't expect much warning of food borne plagues from government agencies!

Another interesting link is that the further you go from the equator, the higher the incidence of Multiple Sclerosis and Lupus.  Also, the further you go from the equator the more the normal diet includes animal protein.  Those who live at the equator or closer to it eat mainly fruit, vegetables, and nuts.

Further, a study has been underway for close to fifty years on MS patients.  According to Doctor Skank, an MS researcher who has now left his practice to another doctor, all of the MS patients he treated with medications and drugs were either dead or wheel chair bound within ten years.  95% of the patients who followed a special diet, no medications, had no progression of the disease in fourty years!  That 'special diet' was a vegan diet.  No animal protein.  No casein, no whey either.  (Casein and whey are dairy products)  

I have heard of a possible link between the Bovine Growth Hormone and the increase in diabetes, too.  It seems the BGH acts upon the natural insulin in humans.  This hormone was given to dairy cattle to promote higher milk production.  Strangly, at the same time they were promoting the use of BGH, the government was paying farmers to cull their dairy herds, slaughter the cattle because of a milk glut!  Does that make any sense??

Title: Missing Link to Alzheimer's?
Post by: Richard Myers on June 06, 2006, 08:07:00 PM
It is being reported that the USDA is saying that the U.S. mad cows had atypical BSE. Well, how about that! Seems to me that there may be two smoking guns.

For those who still don't mind eating animal products, I am sure you are following this closely, at least for your children's and grandchildren's sake.

Title: Missing Link to Alzheimer's?
Post by: Richard Myers on June 06, 2006, 08:09:00 PM
Oh, btw, don't expect to find this on the front page of today's newspaper or on your television network news. I don't think it will be there for some reason.
Title: Missing Link to Alzheimer's?
Post by: Richard Myers on June 06, 2006, 08:47:00 PM
Some are calling this BSE in the US a "new strain". It has a longer incubation time than the mad cow disease in England. 2+2 ummm. You mean that it would take longer to manifest the disease? ummmm. Like instead of getting CJD or maybe Alzheimers at 25 to 35 you would come down at say age 60 or 70?

Very interesting!

Oh, and there is another difference with the American mad cows. It was originally detected through active surveillance of live animals rather than during inspection of a suspect fallen animal. ummm. Maybe slaughtering live cows that don't look sick may be a problem? I guess Japan thinks so since they won't take our beef and they test every single cow they slaughter to see if it has BSE.

For those who have been given great light, it seems to shining ever brighter. We ought to walk in it even as He walked.

Title: Missing Link to Alzheimer's?
Post by: Richard Myers on June 08, 2006, 04:45:00 PM
There is a chinese scientist who has made a very important discovery. He discovered a similar pattern of amino acids in the prion protein and the amyloid precursor protein: a reductive amino acid followed by three non-reductive amino acids. He says "This suggests a common molecular mechanism underlying the initiation stages of sporadic Alzheimer's disease and both sporadic and genetic prion diseases..." Don't you just really see the lie in "sporadic"? It just happens!

Now, it seems that the evidence is building. And, if Alzheimer's is indeed caused by eating infected animal products, then we have a world-wide problem regarding the use of animal products as food. Don't suppose that God's messenger is being confirmed?

When we accept what God has said and use it for the foundation of our studies, we shall be given wisdom that surpasses that of those in the world who reject God and His Word. Does this surprise anyone?

Title: Missing Link to Alzheimer's?
Post by: Richard Myers on June 03, 2007, 09:27:00 AM
We are at the leading edge of science regarding the health of humans. While infectious disease is rampant in the world, few understand the relationship to the injesting of animal products. It is so very sad that God's church has not warned the world, but instead has followed their ways in suggesting a diet composed of animal products is necessary.

Here is another little glimpse into the truth and where we are headed:

At a recent Neuroprion meeting, a study was
presented showing that in transgenic mice BSE passaged in sheep may be more virulent and infectious to a wider range of species than bovine derived BSE.

Other work presented suggested that BSE and bovine amyloidotic spongiform encephalopathy (BASE) MAY BE RELATED. A mutation had been identified in the prion protein gene in an AMERICAN BASE CASE THAT WAS SIMILAR IN NATURE TO A MUTATION FOUND IN CASES OF SPORADIC CJD.

In other words the evidence is building that "sporadic" is not sporadic, but rather infectious from coming in contact with animals, most likely the eating of animals that have a spongiform disease. This more than likely will prove to be the source of Alzheimer's also.

Title: Missing Link to Alzheimer's?
Post by: Mimi on June 04, 2007, 05:48:00 PM
This is big!
Title: Missing Link to Alzheimer's?
Post by: Curt on June 08, 2007, 06:46:00 PM
The scientific community is still baffled about the possible cause of Alzheimers. Today's report points towards a particular Genetic abnormality common in Anzheimer patients which hwas isolated by a computer program on several gene samples.

It is my personal opinion that sometimes the genetic relationships is a case of mapping the result rather than the cause.

Here is today's medical report.

******************************************  
Scientists Find Gene Link to Alzheimer's
June 8, 2007

PHOENIX (AP) -- A team of Arizona researchers think they've found a gene that could help better predict a person's risk of developing Alzheimer's disease.

The gene -- called GAB2 -- seems to affect the odds that some people will get the progressive neurological disease that afflicts about 5 million Americans, according to the research team led by the Translational Genomics Research Institute and Banner Alzheimer's Institute.

"This is a major breakthrough in Alzheimer's genetic research that will have an impact on the clinical treatment of the disease," said Dr. Dietrich Stephan, director of TGen's neurogenomics division.

Researchers here believe the study marks a new milestone for genetic research of Alzheimer's disease because it used a high-powered computer chip to measure more than a half-million genetic variations, the most robust such study to date.

Alzheimer's triggers memory lapses, clouds the thought process and leads to confusion and death in older adults.

About 78,000 Arizonans suffered from Alzheimer's in 2000, a number expected to jump to 130,000 by 2025, according to Banner Alzheimer's Institute.

Researchers worldwide are not sure what causes the disease. They do know that sufferers' brains are harmed by plaques and tangles that block signals and ultimately cause cells to shrink and die.

TGen's Stephan began investigating the possibility of conducting an ambitious study of the disease three years ago.

He sought funding from the Kronos Science Laboratory in Phoenix, which provided most of the money for the $5 million project.

In turn, Kronos secured the intellectual property rights from the Arizona study and is seeking patent protection for the GAB2 gene and its role in the onset of Alzheimer's.

The study is the latest to draw national attention for the gene investigators at TGen, which launched here five years ago as part of a push to build Arizona's research prowess.

Other significant studies conducted by TGen and collaborators in Arizona include genetic tests relating to pancreatic cancer and amyotrophic lateral sclerosis, also known as Lou Gehrig's disease.

Copyright 2007 The Associated Press. All rights reserved. This material may not be published, broadcast, rewritten or redistributed.

Title: Missing Link to Alzheimer's?
Post by: Richard Myers on June 08, 2007, 09:43:00 PM
I think that genetics plays a part in the ease with which disease may find a foothold. But, it is an excuse to a large degree for poor lifestyle. Drink alcohol and you will suffer. Some more than others. The solution is not to use stem cells, but to refuse to drink.
Title: Missing Link to Alzheimer's?
Post by: Curt on June 12, 2007, 06:20:00 AM
New research out today shows that treating the Heart related symptoms can assist in controlling the degeneration caused by Alzeimhers. So, even a non-drug approach to controlling high blood pressure and cholesterol can help.

*********************************
   
Heart Disease Treatments May Slow Alzheimer's
June 11, 2007

(USA TODAY) -- Treatment of cardiovascular risk factors such as high blood pressure might slow the progression of Alzheimer's, the incurable brain disease that causes memory loss and confusion in millions of people.

That's the conclusion from a study presented Sunday at the second Alzheimer's Association International Conference on the Prevention of Dementia in Washington, D.C. The findings add to growing evidence suggesting that reducing the risk of heart disease might also shield the brain from a disease that will skyrocket in the coming decades.

If nothing is done to stop the explosion of cases, Alzheimer's worldwide will quadruple from 26.6 million to more than 100 million by 2050, according to a second study presented at the meeting by Ron Brookmeyer, a researcher at the Johns Hopkins Bloomberg School of Public Health.

Previous research had suggested that treatment of high cholesterol and high blood pressure might help prevent Alzheimer's in healthy people. A study at the meeting now suggests that such treatment might actually slow the progression of established disease.

Yan Deschaintre of the Memory Center in Lille, France, studied 891 dementia patients, including people with Alzheimer's. His team gave patients a test that measures thinking ability at the start of the study and then again four years later. The team noted whether patients got treatment during that four-year period for high blood pressure, high blood cholesterol or diabetes.

Patients who received treatment did better on the final thinking and memory test than the people who got no care.

Some doctors don't bother to treat high blood pressure or other cardiovascular risk factors in such cases because they think the treatment won't make much difference for a patient who's declining rapidly, Deschaintre says. But the new findings suggest even standard care for heart risk factors, like reducing high blood cholesterol with a drug, might help an Alzheimer's patient think a little more clearly, Deschaintre says.

A third study, also presented at the meeting, suggests that optimism and the ability to establish and carry out goals might be traits that help keep the aging brain in shape. Simon Forstmeier and colleagues at the University of Zurich in Switzerland studied 120 people ages 60 to 95. He found that seniors with a can-do attitude did better on memory and thinking tests given at the time of the study.

Forstmeier says people can be trained to be more optimistic, to regulate emotion, and to formulate and achieve goals -- all traits that might help build a brain resistant to Alzheimer's.

"We don't know if we'll be able to fully prevent Alzheimer's," Brookmeyer says. But he and other experts are urging people to take steps now that might reduce their risk: "Even a small delay in the disease will have a big impact."

Copyright 2007 USA TODAY, a division of Gannett Co. Inc.

Title: Missing Link to Alzheimer's?
Post by: Richard Myers on September 13, 2007, 09:35:00 PM
Here we go, another little piece of the puzzle.  

"Amid growing evidence that the same abnormal clumping of proteins in Alzheimer's disease also contributes to type-2 diabetes, scientists in New York are reporting discovery of a potent new compound that reduces formation of those so-called amyloid plaques.

The report cites evidence correlating increases in amyloid formation in the pancreas with increases in severity and rate of progression of type-2 diabetes, which affects almost 20 million Americans and is rapidly rising worldwide.

Deposits of the abnormal protein damage and destroy insulin-producing "islet" cells in the pancreas. Researchers have been seeking potential new medicines that block formation of an abnormal, misfolded protein called islet amyloid polypeptide (IAPP), which may play a key role in the cell destruction."

Ever hear of a "misfolded protein"? That is what a prion is. Prions are what cause Mad Cow Disease in cows, sheep, deer, cats, and humans.  Now we that amyloid plaques are being related to "misfolded proteins".  Don't we just love mysteries as they unfold! We were warned to avoid animal products for this very reason.  

I don't know about diabetes and misfolded proteins, but I surely would like to think that we can prevent Alzheimer's by eating a strict vegetarian diet.  BTW....we have cured diabetics (type 2) with a strict vegetarian diet and exercise program.

Title: Re: Missing Link to Alzheimer's?
Post by: Richard Myers on March 24, 2008, 09:03:55 AM
Here is some information on alzheimer's if one is interested in how widespread the disease is and much it costs society.
source (http://www.alzheimersanddementia.org/webfiles/images/journals/jalz/JALZ_739.pdf)
Title: Re: Missing Link to Alzheimer's?
Post by: Richard Myers on March 24, 2008, 09:50:16 AM
15 years ago, I did not find one who would say that Alzheimer's was connected to Mad Cow or Scrapie
Disease. Today, we are seeing research findings that are indicating a relationship. God has blessed His people with light in advance of what science has discovered.

      Association between Deposition of Beta-Amyloid and Pathological Prion Protein in Sporadic Creutzfeldt-Jakob Disease
Debatin, L. ;    Streffer, J. ;    Geissen, M. ;    Matschke, J. ;    Aguzzi, A. ;    Glatzel, M.
Neurodegenerative Dis (DOI:10.1159/000121389)
Published Online: March 18, 2008    

Original Paper

Association between Deposition of Beta-Amyloid and Pathological Prion Protein in Sporadic Creutzfeldt-Jakob Disease
Laura Debatina, Johannes Strefferb, Markus Geissenc, Jakob Matschkec, Adriano Aguzzia, Markus Glatzela, c

aInstitute of Neuropathology, and
bDivision of Psychiatry Research, University Hospital Zurich, Zurich, Switzerland;
cInstitute of Neuropathology, University Medical Center Hamburg-Eppendorf, Hamburg, Germany

Address of Corresponding Author

Neurodegenerative Dis (DOI: 10.1159/000121389)

 goto top of page Key Words

    * Sporadic Creutzfeldt-Jakob disease
    * Alzheimer's disease
    * Deposition of beta-amyloid
    * Prion protein

 goto top of page Abstract

Background: Alzheimer's disease (AD) and prion diseases such as sporadic Creutzfeldt-Jakob disease (sCJD) share common features concerning their molecular pathogenesis and neuropathological presentation and the coexistence of AD and CJD in patients suggest an association between the deposition of the proteolytically processed form of the amyloid precursor protein, beta-amyloid (Abeta), which deposits in AD, and the abnormal form of the prion protein, PrPSc, which deposits in sCJD. Methods: We have characterized sCJD patients (n = 14), AD patients (n = 5) and nondemented controls (n = 5) with respect to the deposition of PrPSc and Abeta morphologically, biochemically and genetically and correlated these findings to clinical data. Results: sCJD-diseased individuals with abundant deposits of Abeta present with a specific clinicopathological profile, defined by higher age at disease onset, long disease duration, a genetic profile and only minimal amounts of PrPSc in the cerebellum. Conclusion: The co-occurrence of pathological changes typical for sCJD and AD in combination with the inverse association between accumulation of Abeta and PrPSc in a subgroup of sCJD patients is indicative of common pathways involved in the generation or clearance of Abeta and PrPSc in a subgroup of sCJD patients.

What has been seen? Look at the conclusion. I have been pointing to the amyloid plaques that are seen in Alzheimer's brains and the deposition of the same type of material in Scrapie and now in some "mad cows". This study has revealed that the  Abeta (amyloid plaque), and the PrPSc (prion protien) appear to share a common route of infection. Science is catching up with the light we have been given. Alzheimers is not  caused by aluminum. And, sporadic CJD does not just happen. There is nothing in this study to suggest that mad cows or mad sheep cause either sCJD or Alzheimer's, but we are getting very close to knowing through science that this is the case. We know that mad cows infect humans and cause vCJD. We now know that there is a connection between Alzheimer's and sCJD. But, the meat and dairy industry does not want you to know that both Alzheimer's and sCJD are being transmitted to humans via infected animals, both sheep and cows (and probably other animals that may be carriers). Pigs and chickens are fed material that comes from downer cows that cannot be fed to cows and sheep.

It is not a stretch to understand that sporadic CJD is an infection with a cause. What cause? The same as variant CJD, infected meat. When this is understood, the study we have just been reviewing makes it clear that if sCJD is from infected meat, then since both sCJD and Alzheimer's share a "common pathway" for the development of both the amyloid plaque and the prions, we would then have our connection between mad cows or mad sheep and Alzheimer's.

How long before the evidence is clear? I think it already is. The new varieties, strains, of Mad Cow that we are seeing in Europe and the US which produce the amyloid plaques and the fact that these amyloid plaques have been seen in Scrapie sheep is enough evidence for those who are not biased to say there is reason to believe there is a connection. In fact what we will probably find is that Alzheimer's is in fact Scrapie or Mad Cow Disease in humans. Yes, there are different manifestations of the infection, but the cause is the same, infected meat in which cooking does not inhibit the transmission of the disease.
Title: Re: Missing Link to Alzheimer's?
Post by: Mimi on March 24, 2008, 11:16:00 AM
Incredible information. Everything we have seen relative to this has been leading up to these findings.
Title: Re: Missing Link to Alzheimer's?
Post by: Richard Myers on March 24, 2008, 06:06:49 PM
This is a fatal disease. No cure. And, do not listen to the "experts" say again that they do not sell that part of the animal that transmits Mad Cow Disease. It is very probable that other than specified risk material carries the infected prions. And, this may include milk. There is no safety in eating other than the original diet given to man. A plant based diet free from all animal products. This is the healthiest as well as the safest.
Title: Re: Missing Link to Alzheimer's?
Post by: Richard Myers on March 24, 2008, 06:08:18 PM
I have a request of our readers. If you know of any other Seventh-day Adventists or even non SDAs that are discussing this or ever have, please let me know. Thank you.
Title: Re: Missing Link to Alzheimer's?
Post by: Mimi on March 25, 2008, 03:04:36 AM
Using various key search words, I gathered up some info and sent it to you. It appears there are those in the field who have been looking into this as far back as 1987.
Title: Re: Missing Link to Alzheimer's?
Post by: Richard Myers on March 25, 2008, 01:12:57 PM
Thank you. As I have time, I will post the associations that we can see are known.

From (Seventh-day) Adventist Health:

Researchers are also investigating environmental factors (infections, metals, industrial and other toxins) that may trigger oxidation, inflammation, and the disease process, particularly in people with a genetic susceptibility to Alzheimer's.

Infectious Organisms. Slow, infectious viruses cause a number of other degenerative neurologic diseases, such as kuru and Creutzfeldt-Jakob disease.
Creuztfeldt-Jakob disease

Although no specific virus has been linked to Alzheimer's, some researchers theorize that people with a genetic susceptibility to Alzheimer's may be vulnerable to the actions of certain viruses, particularly under circumstances when the immune system may be weakened.

No connection is made here with BSE. Only a thought that some researchers somewhere "theorize" that some people may be susceptible to certain viruses. They only mention CJD as an example of a disease that is caused by a slow virus. At least they did mention that there are some that theorize a virus may cause the disease. 
Title: Re: Missing Link to Alzheimer's?
Post by: Richard Myers on March 26, 2008, 12:33:32 PM
Loma Linda University Receives Grant for Alzheimer's Disease Research

A recent $6.55 million grant from the National Institutes of Health to Loma Linda University School of Medicine's Neurosurgery Center for Research, Training and Education will provide means for research into a cure for Alzheimer's disease.

January 7, 2003 Loma Linda, California, United States


A recent $6.55 million grant from the National Institutes of Health to Loma Linda University School of Medicine's Neurosurgery Center for Research, Training and Education will provide means for research into a cure for Alzheimer's disease.

The disease, which researchers call "deadly," is the eighth-leading cause of death in the United States, according to the Centers for Disease Control and Prevention.

Researchers at LLU School of Medicine believe there is a cure for AD, but they must first determine what causes the disease. The research team plans to develop a diagnostic tool for early diagnosis of AD.

Commercially available screening tests for Alzheimer's may not be too far away, researchers believe. Initial screening clinics, part of a five-year study sponsored by the Alzheimer's Association, has already begun and will ultimately include 100 healthy seniors 50 years of age and older who live in southern California.

"My feeling is that other researchers are going to get into this," says Cindy Dickson, administrator for LLU School of Medicine. "I think that there's going to be a spin-off research. They're going to see something that might attract them to what we're doing here."

"I am excited about the project because of its comprehensive design," says Lora Green, Ph.D., associate professor of microbiology and the radiobiology program at LLU School of Medicine. "And even if all of our goals are not met, we will have made significant roads into better understanding the biological processes of Alzheimer's disease and thereby improving the diagnostic options and potential for earlier therapeutic intervention."

Loma Linda University Medical Center, established by the Adventist Church in 1905, is internationally renowned for its medical research and treatments in areas such as heart transplant surgery and non-invasive proton beam therapy for prostate and breast cancer.

Source: (Seventh-day) Adventist News Network
Title: Re: Missing Link to Alzheimer's?
Post by: Richard Myers on March 26, 2008, 12:45:41 PM
Well....in 2003 LLU did not seem to have any idea that they may be dealing with a relationship to CJD. They are looking for a cure? And, I suppose a cure for CJD also?   We have enough light to prevent Alzhemier's. The $6,500,000 could be better spent in preventing Alzheimer's. And, that includes showing that this infectious disease most likely is coming from eating animals products.
Title: Re: Missing Link to Alzheimer's?
Post by: Mimi on March 26, 2008, 01:27:42 PM
Maybe you need to call a meeting and present your evidence. There is certainly enough of it just on this forum alone.

Title: Re: Missing Link to Alzheimer's?
Post by: Richard Myers on March 26, 2008, 08:46:48 PM
Call a meeting?  Whom shall we invite? Those who want to know what causes Alzheimer's and then will walk in the light will make good Christians. :)  We are calling them!  :) And the meeting?  The marriage supper of the Lamb!  :)
Title: Re: Missing Link to Alzheimer's?
Post by: stephen on March 27, 2008, 08:59:17 PM
Amen.

At a small Church here in Central Michigan (Cadillac), there are more baptisms, per capita, than any of the other Churches I know of.  It is obviously due to the STARTNEW program.  Now, there are Churches in Grand Rapids, Muskegon area, Grayling, West Branch, Manton, and others who are piloting the program.  Soon to join is Reed City and Big Rapids.  The reason for the success of STARTNEW?  Truth.  With a Capitol T.  God's word is not minced here.  His eight laws of health are addressed without reservation.  Our 'Councils on Diet and Health' are believed and expounded upon.  The use of dairy products and eggs are also discouraged along with flesh meats.  The rise of disease and food borne plagues is openly discussed. 

Some in our own conference consider this program to be 'divisive' because some Seventh Day Adventists do not wish to 'give up' their dainties.  They prefer 'smooth things'.  It is interesting how those outside our own Church are more accepting of our health message than many of our own members!  And as stated above, the result has been in baptisms.  Praise God.

But the truth must be told.  Thank you, Brother Richard, for your investigative work on these diseases and your commitment to raising the flag concerning them.
Title: Re: Missing Link to Alzheimer's?
Post by: Larry Lyons on March 28, 2008, 09:54:31 PM
Richard Rhodes wrote a book "Deadly Feasts" which documented the search and ultimate discovery of the cause of Kuru and its link to the related diseases. He had interviews with the original researcher, an American physician who was connected with the CDC. The doctor had travelled back and forth to New Guinea with vials of tissue and blood in his shirt pocket, (imagine trying that today) and in an interview warned that there could be an epidemic of TSE that would affect hundreds of thousands of people. It is well worth reading if you haven't read it.

In his book he tells of one of the first cases, a young teenage girl in England, whose death was acknowleged to be related to eating infected meat. An official from the government visited the parents of the girl and encouraged them to not tell anyone it was from eating beef. "Think of the effect on the economy if this gets out," he told them.
Title: Re: Missing Link to Alzheimer's?
Post by: Richard Myers on March 28, 2008, 10:22:24 PM
It is interesting that you bring up Kuru, Brother Larry. While it may have been difficult to draw a connection between Kuru and CJD or Kuru and BSE, there ought to have been a real concern about Scrapie sheep and CJD. And, today, we hear nothing, absolutley nothing about Scrapie being a risk for CJD. Did you read about CJD and its connection to sheep?
Title: Re: Missing Link to Alzheimer's?
Post by: Larry Lyons on March 29, 2008, 08:27:54 AM
Richard, yes, it is brought out in the book that scrapie infected sheep have similar spongiform lesions as do the other related diseases. I don't have a copy of the book now (loaned it to some one) but I seem to recall that the cause of scrapie was unknown and has been around for a very long time. The sheep were not feed animal by products. I noticed when I googled "Deadly Feasts" that it gave 2000 as the published date. I'm pretty sure it came out before 1997 so it may hae been up dated.
Title: Re: Missing Link to Alzheimer's?
Post by: Richard Myers on March 29, 2008, 04:52:41 PM
The information I am going to share is not widely published. It is not hidden, but neither is it shared. I have purposely not posted it in the more than eight years we have been online. I wanted to see if anyone knew about it. Few do. How important is the information? I believe it is very important and when Seventh-day Adventists who are walking in the light read it, they will understand. Most will go on with life as if they never read it. They will continue to eat animal products and feed their children likewise as if it were safe to do so.

Here is the statement--

The largest cluster of CJD occurs among Libyan Jews, where the incidence (1/10,000) is ∼100 times higher than incidence worldwide. The origin of the higher incidence of CJD in this population is an intriguing problem that has not yet been resolved.

Although a few studies (reviewed in Meiner et al. 1997) had pointed out the familial predisposition to CJD, it was first speculated that its frequency in Libyan Jews could be explained by their habit of consuming lightly grilled sheep’s brains or eyeballs (Herzberg et al. 1974; Alter and Kahana 1976), reflecting a shared environmental risk (exposure to scrapie-infected meat) rather than any genetic factor.

While there does appear to be a genetic factor in this disease, there is also an infectious transmission of the disease. The brain has the highest concentration of infected prions and the eyeball is connected to the brain. It would make no difference as to the cooking time, the material is highly infectious.

This truth regarding CJD, the human from of Scrapie and mad cow disease, is something that the human population has not been told. That the highest concentration of humans with CJD occurs in a population that eats sheep's brains and eyeballs is not something that can be dismissed as unimportant. This human form of Scrapie is complex. Not all who eat it come down with it. Not all who drink milk come down with Leukemia, but when the infectious agent is eaten, it puts one at risk for these infectious diseases. God has given to man a wondrous immune system, but He asks us to work with Him in preventing disease. When it comes to us that disease is being transmitted through the eating of diseased animals and their milk and eggs, it is only common sense to to understand that one is at risk by eating animal products.

If we had to eat animal products to be healthy, we would be in a spot. But, we do not. The healthiest people I know are third generation strict vegetarians. Eating a Bible based plant diet will not only remove the risk posed by diseased animals, but it will provide the best nutrition that the human body requires.
Title: Re: Missing Link to Alzheimer's?
Post by: Suzanne on July 22, 2008, 03:30:59 PM
I am in the process of reading the book: "Dying For A Hamburger, Modern Meat Processing and the Epidemic of Alzheimer's Disease, by Murray Waldman, MD, and Marjorie Lamb. Here the authors discuss kuru, scrapie, CJD, etc.  While I have not as yet finished the book, its quite interesting how they show that Alzheimer's Disease is a fairly new phenomenon, tying it in with modern meat processing and eating.  I am just now coming to part 3 of the book where it discusses Prion Diseases.

When I finish I can give a complete report--its quite informative and interesting so far.

Also, I read and have the book: Deadly Feasts, - Tracking the Secrets of a Terrifying New Plague,  by Richard Rhodes. I will have to look through it again to give a better report. (I read it several years ago). Leafing through the book I found these excerpts underlined in red: "How the new disease (CJD) spreads is known:  it spreads in the cannibalism of animals by animals, it spreads in the industrial cannibalism of animal remains fed to animals, it spreads by the eating of beef."

As mentioned in a prior post, it discusses kuru, scrapie, CJD, etc.  It also discusses multiple sclerosis and Alzheimer's, diseases of the central nervous system. In chapter 11, Meat Bites Back, Mad Cow Disease is discussed, tying it in with kuru.  One researcher noted that mad cow disease is kuru and nothing but kuru, and "any species could be carrying it--dairy cows, beef cattle, pigs, chickens...."

...quite interesting, will have to reread the book.

Suzanne   
Title: Re: Missing Link to Alzheimer's?
Post by: Suzanne on July 23, 2008, 03:31:11 PM
Dr. Mehmet Oz, the celebrated heart surgeon, who emphasizes natural health measures was on Oprah yesterday (7-22-08). They covered several subject one of which is that the country of India has the least amount of Alzheimer's patients of any country.  They noted that they use colorful turmeric in so much of their food, which could be a factor in this phenomenon. 

I couldn'd help but think that the fact that they do not eat beef figures heavily in the low incidence of Alzheimer's there. 

Dr. Oz says the real role of a physician is to educate and guide patients as they seek to live their most healthful lives.

Suzanne
Title: Re: Missing Link to Alzheimer's?
Post by: Richard Myers on July 26, 2008, 12:35:34 PM
Yes, Dr. Oz does not have the light that we have been given, so he makes a wrong assessment of the cause of Alzheimer's. We are truly blessed to be able to draw correct conclusion from such observations. Soon, the world will see the cause and they then will see that we were correct. That God does care about them and did in fact warn them.

For those who may not know, cows are holy in India, so they are not eaten by the Hindus. Cow and bull worship was a common practice in many parts of the world, beginning in Mesopotamia around 6,000 B.C. and spreading to Northwestern India with the invasion of the Indus Valley in the second millennium B.C. by Aryan nomadic pastoralists who established the Vedic religion. What is remarkable is that such worship has persisted uniquely in India to the present day. Lodrick (1981) concludes that revulsion against sacrifice, the economic usefulness of cattle and religious symbolism ( especially as the Mother-provider) were factors contributing to the formulation of the sacred cow doctrine.  Anti-Biblical.
Title: Re: Missing Link to Alzheimer's?
Post by: LindaRS on July 26, 2008, 04:20:42 PM
I see Larry has already mentioned the book Deadly Feasts. I read the book several years ago. One thing in the book stood out clearly to me—that CJD (prion) disease have been in this country (the USA) for many years. It has affected many different kinds of animals, and if I recall correctly, was found in chinchillas (or it may have been minks) that are raised for their fur which is made into coats and such. Those who have an interest in BSE will find the book most interesting and enlightening.
Title: Re: Missing Link to Alzheimer's?
Post by: Richard Myers on July 26, 2008, 04:29:28 PM
Yes, spongiform disease was found in minks. They had been fed cow meat.  This was found in the mink before the US said they discovered BSE in US cattle.
Title: Re: Missing Link to Alzheimer's?
Post by: Richard Myers on March 10, 2010, 09:18:02 AM
Here's some bad information for Canadians. The number of cases of Alzheimer's is expected to increase dramatically in Canada. source (http://www.alzheimer.ca/docs/RisingTide/AS%20Rising%20Tide-Executive%20Summary_Eng_FINAL_SecuredVersion.pdf)

In case citizens of other countries think they are safe, I don't think so. If you eat animals or animal products, it appears that you may be at risk as the incidence of dementia is increasing world-wide.
Title: Re: Missing Link to Alzheimer's?
Post by: Richard Myers on May 12, 2010, 11:58:20 AM
This morning, I have been doing some research in this area. It is my belief that Alzheimer's is an infectious disease. I believe it to be directly related to spongiform disease in animals and man and have expressed this thought many times in this thread. There is evidence in the literature to suggest this. My understanding is not based just on my study of the light we have been given regarding the connection between the disease in animals and man, but rather on both the inspired counsel we have and the scientific literature published.

Here is an excerpt from an exciting article that may help others to see the truth from my perspective:

Is AA amyloidosis in cheetahs a prion disease? The answer depends on whether AA amyloidosis in captive cheetahs is caused by spontaneous disease or transmission of amyloid between animals. Environmental influences on AA amyloidosis epidemiology could be due to the presence of either “infectious” amyloid, a prion-like etiology, or to factors that enhance the incidence of spontaneous disease, i.e., a non-prion etiology. Even if transfer of AA amyloid between cheetahs enhances AA amyloidosis, the question would remain as to whether the transferred amyloid initiates the disease de novo or merely accelerates ongoing disease. The latter scenario would place AA amyloidosis into a gray area with respect to the basic prion concept. In this instance, prion transmission would affect the kinetics of the disease without actually initiating it.

Regardless of prion semantics, there could be practical consequences of such kinetic phenomena in both animals and humans. For instance, recent studies have shown that injection of β-amyloid can enhance Alzheimer's-like amyloidosis in transgenic mice (14). This raises the possibility that inadvertent transfer of β-amyloid from one person to another could accelerate the neurodegenerative process to the point where it becomes Alzheimer's disease as opposed to normal aging. In this example, as well as in cheetah AA amyloidosis and many other protein misfolding diseases, the basic problem is likely the outpacing of an organism's protein quality control mechanisms. This may sometimes be more a problem of the rate, rather than of the instigation, of protein misfolding. source (http://www.ncbi.nlm.nih.gov/pmc/articles/PMC2438212/)

What we see here is that there are some that are beginning to believe that Alzheimer's may be infectious. And....that the infection source can be other "prion" type disease infected animals via feces or by ingestion of the carcass of the dead animal. And, it has been shown that milk is a vector for the transmission of some prion diseases.

When we consider that we have had Scrapie and Mad Cows for some time, it becomes a matter of how great is the risk from eating animal products? Well....if what I believe about the relationship between Alzheimer's and animal products is true, then one need merely count the cases of Alzheimer's. And, if that does not speak to risk, then consider that the numbers are increasing and is projected to continue to increase.

The threat of cancer from eating animals and their milk and eggs does not seem to concern some, but there are worse things to die from. The mind is a precious gift and when it begins to be eaten by CJD or affected by amyloid plaques it is a fearful thing. Better to prevent the disease.
Title: Re: Missing Link to Alzheimer's?
Post by: Geodad on May 13, 2010, 12:11:46 PM
Interesting information. I recall another recent news article indicating that having a spouse with Alzheimer's increases one's risk of the disease 600%. If it is infectious, hopefully there is not significant a transmissive risk to other members of the household just from providing care to a elderly parent.
Title: Re: Missing Link to Alzheimer's?
Post by: Richard Myers on May 13, 2010, 05:04:30 PM
I  don't think  it is a great danger from such contact. Like a slow retro virus, it appears to not be contracted through such contacts. It is hard to know because the incubation period is usually so long. The infectious agent is much more prevalent in nerve tissue. The eye and brain are especially loaded with infectious agents. There also appears to be a genetic makeup that makes one more susceptible to contracting the disease.
Title: Re: Missing Link to Alzheimer's?
Post by: colporteur on July 14, 2010, 06:24:06 PM
I have heard that alzheiner's victims have aluminum in the brain. Is this true ?
Title: Re: Missing Link to Alzheimer's?
Post by: Larry Lyons on July 14, 2010, 06:52:57 PM

Yes, but there are pro and con opinions about it. I heard about the dangers of using aluminum cookware decades ago. If I remember right, Jethro Kloss talks about it in "Back to Eden."
Title: Re: Missing Link to Alzheimer's?
Post by: colporteur on July 15, 2010, 07:45:44 AM
My intertest in this has to do with canvassing. Typically with a health interest I will begin with, to peak interest, that everyone that dies of alzheimers has aluminum in the brain. I have heard this. Then I state that there is a place where people get aluminum from that they know nothing about, that being baking powder. Sometimes people will dig out their baking powder to see. An ingredient listed on the label is aluminum sulfate. The solution is to go to Walmart and purchase aluminum free baking powder. While even the aluminum free powder may not be the best solution at least it does not have aluminum. Then I state how and why dairy being high protein tends more to make weak bones than strong bones and you have the person's interest.

I don't think they hardly every autopsy an Alz. corpse but have wondered if the existance of aluminum is sometimes. often, or always in the brain.
Title: Re: Missing Link to Alzheimer's?
Post by: Curt on October 26, 2010, 06:25:59 PM
There are many documented connections to Alzeimer's. More on Alzheimer's

Smoking Raises Alzheimer's Risk
(USA TODAY) -- Heavy smoking in midlife more than doubles your odds of developing Alzheimer's disease, a Kaiser Permanente study said Monday.

Phoenix Arizona, June 8th 2007. Translational Research Institute and Banner's Alzheimers Institute
The gene called GAB2 seems to affect the odds of a person developing the progressive neurological disease.

May 3rd 2006 Washingto (AP) - New study shows a link between Diabetes and Alzheimers. Alzheimers can be easily triggered when brain cells cannot properly use their main food, sugar. "When they are in and insulin resistant state it not only affects the body, it affects the brain as well."

Harvard Medical School - Vitamin B12 may lower risks of Alzheimers
Patients in the study group with high levels of homocyceine (protein building block) developed Alzheimers. the active component of B12 helps to lower homocysteine levels.

Jenny Hope, Daily Mail, UK, June 4th 2009
Cumin prevents the spread of amyloid plaques in the brain of Alzheimers patients. The plaque is thought to play a part in memory loss and mental impairment.



Title: Re: Missing Link to Alzheimer's?
Post by: Richard Myers on February 01, 2012, 11:38:10 PM
I don't have time to spend doing research on this subject, but I took some time tonight ( it is late) to take a look at what has been happening.  I don't have to express my joy at finding this because those who have been following this thread

Alzheimer's disease: A prion protein connection

Moustapha Cisse1 & Lennart Mucke1

February 26, 2009

More than 20 million people worldwide have Alzheimer's disease, yet its causes remain mostly uncertain. Fresh findings provide molecular clues, linking this disease to another neurodegenerative disorder.

Investigations of the causes of Alzheimer's disease yield one culprit time and time again: abnormal build-up of amyloid-β (Aβ) peptides in the brain. Small, soluble aggregates of Aβ — Aβ oligomers — impair memory by disrupting memory-related functions of synaptic junctions between neurons1, 2, 3.  source (http://www.nature.com/nature/journal/v457/n7233/full/4571090a.html)
Title: Re: Missing Link to Alzheimer's?
Post by: Geodad on February 02, 2012, 07:43:21 AM
Apparently some new light has been shown on a possible Alzheimer's mechanism. See link below. It appears there were various theories about how the damage to brain tissue took place. One was that the beta-amyloid deposits outside the neurons created a "bad neighborhood" conducive to cell damage and death and that the least resistant cells were the first to die. Another theory was that something was spreading from cell to cell. That something appears to be another protein called tau. Probably the real disease involves some type of synergy between the two processes...

While I would like to think it improves one's chances, I am not sure that an all-plant vegetarian/vegan diet will necessarily prevent Alzheimer's. I've seen Alzheimer's patients who had been vegetarians for many decades...on the other hand the prion incubation period can be decades or more also, so I could be mistaken...

http://www.nytimes.com/2012/02/02/health/research/alzheimers-spreads-like-a-virus-in-the-brain-studies-find.html?_r=1&nl=todaysheadlines&emc=tha2 (http://www.nytimes.com/2012/02/02/health/research/alzheimers-spreads-like-a-virus-in-the-brain-studies-find.html?_r=1&nl=todaysheadlines&emc=tha2)
Title: Re: Missing Link to Alzheimer's?
Post by: Richard Myers on February 02, 2012, 09:24:32 AM
I too, have considered how it is that a strict vegetarian could become infected with a prion disease.  First, vegetarian is given by many to mean one who eats more than vegetables. Interesting, but it is common usage of those who still eat animal products and even fish.  It seems that if  one  ceases eating the carcasses of mammals they are "vegetarians". 

The transfer of prion disease  can be through more than eating the flesh of an infected animal. Thus, a "vegetarian" places themselves at risk.  My concern was with a strict vegetarian.  Sadly, they often are placed at risk without knowing it.   One who is trying very hard to do right, may take an herb that is in a capsule made of gelatin.  This is a high risk material for prion disease.  Especially when the source was being imported from the UK.  The manufacturers said that it would not transmit prion disease, but they later said they were wrong.   The process of making the gelatin did not render the material safe.

We can go on and list a number of ways that a strict vegetarian can be infected, but we have done this in the various topics on CJD, Mad Cow Disease and People, etc.  Dental treatment, blood transfusions. surgical instruments,  medicines derived from blood and urine, etc.  Because a strict vegetarian is infected is not evidence that Alzheimers is not related to prion disease.  The evidence suggests that there may be a direct link to prion disease.
Title: Re: Missing Link to Alzheimer's?
Post by: Richard Myers on February 05, 2012, 08:39:33 PM
"Furthermore, we provide evidence that PrPc is expressed in endothelial cells and, that monomeric Aβ1−40 binds to PrPc. These observations provide new mechanistic insights into the role of PrPc (prion protein) in AD (Alzheimer's disease)."  This is up to date research on the subject. An Abstract of article published in  Journal of Cerebral Blood Flow & Metabolism , (1 February 2012) | doi:10.1038/jcbfm.2012.7

PrPc, prions, are associated with Mad Cow Disease (BSE), Mad Deer and Elk Disease (CWD), Mad Sheep and Goat Disease (Scrapie), and others.  Now the evidence increases that there is an association with Alzheimer's Disease. Warnings have been given and ignored. Many do not wish to know that what they are eating and feeding their families is putting them at risk for fatal diseases such as CJD, Cancer, and Alzheimer's.  Alzheimer's is on the increase and is an epidemic in the world as is cancer.  When diagnosis is made, then a cure is hoped for.  There is none. But, prevention is possible. The eating of animal products is the cause of many infectious diseases. The healthiest people are third generation strict vegetarians.
Title: Re: Missing Link to Alzheimer's?
Post by: Richard Myers on August 03, 2012, 03:08:55 PM
Sooner or later science discovers uncovered truth.  The warning we have been giving regarding eating animal products will soon be seen to be real.  Today I found encouraging news that others are beginning to suspect that Alzheimer's may be an infectious disease.  The Scottish TSE Network is advertising a seminar to be held the 12th of November at The Roslin Institute. Title of the seminar "Is Alzheimer's Disease a transmissible disease?"   source (http://www.stn.ed.ac.uk/stn/seminars.html)

God loves us and has not kept us ignorant of such dangers. Science is discovering more and more, yet the eating of animal products continues, even by those who ought to know better.

Title: Re: Missing Link to Alzheimer's?
Post by: Richard Myers on March 13, 2013, 10:05:28 AM
New research is shedding light on the spread of Mad Cow type diseases. It seems that the different varieties of spongiform diseases may come about as they pass through different species of animals and humans. They appear to "adapt" and are able to cross specie boundaries as they adapt. abibleanswer.org (http://abibleanswer.org/index.php?option=com_content&view=article&id=489:mad-cow-type-diseases-spread&catid=105:medical-studies&Itemid=157)
Title: Re: Missing Link to Alzheimer's?
Post by: Richard Myers on May 04, 2013, 06:48:54 AM
There are others who are beginning to be concerned about meat and dairy and a possible relationship to Alzheimer's.

"All of this brings up the unthinkable: that Alzheimer's, Cruetzfeldt-Jackob, and Mad Cow Disease might just be caused by eating the meat or dairy in consumer products or feed. It is only appropriate therefore to explore the role of bovine TB and the atypical mycobacteria in Alzheimer's, JCD and Mad Cow disease and develop better serological surveillance for these pathogens."  National Institute of Health (http://www.ncbi.nlm.nih.gov/pubmed/15694685)
Title: Re: Missing Link to Alzheimer's?
Post by: Mimi on May 04, 2013, 01:13:28 PM
And that was a 2005 statement.
Title: Re: Missing Link to Alzheimer's?
Post by: Richard Myers on May 04, 2013, 01:26:22 PM
Yes, the world is slow to respond. Still most see no relationship between Alzheimer's and what they eat...unless it has aluminum in it. :(
Title: Re: Missing Link to Alzheimer's?
Post by: Richard Myers on September 09, 2014, 07:44:26 AM
For Seventh-day Adventists who have had great light and not walked in it, they have put themselves in a high risk position. While the Alzheimer's epidemic continues to grow, the evidence mounts suggesting it is infectious and associated with prion disease. If true, this puts all who consume animal products at risk.

In May of this year we find published this interesting statement about the connection between prion disease and Alzheimer's:
"Mounting evidence argues that prions feature in the pathogenesis of many, if not all, neurodegenerative diseases. Such disorders include Alzheimer’s, Parkinson’s, Lou Gehrig’s and Creutzfeldt-Jakob diseases as well as the frontotemporal dementias. In each of these illnesses, aberrant forms of a particular protein accumulate as pathological deposits referred to as amyloid plaques, neurofibrillary tangles, Lewy bodies, as well as glial cytoplasmic and/or nuclear inclusions." University of California, institute for neurodegenerative Diseases (https://www.landesbioscience.com/journals/prion/2.Plenary%20session_abstracts.pdf)
Title: Re: Missing Link to Alzheimer's?
Post by: colporteur on September 09, 2014, 08:39:05 AM
 Alzheimer's is just a label that really does not in itself mean anything. Just like cancer there may be more than one brain ailment tagged Alzheimer's. Brains that have been labeled such are often found to contain a considerable amount of mercury, aluminum and other metals. Cheese has been found to be the #1 source  of aluminum in the body. Either way, Alzheimer's is directly linked to animal products of one kind or another.

When God said move  away from animal products especially meat and cheese He meant it. While the world may have a little less light on this particularly since animal product industries are trying to peddle their product  and therefore speak false information, SDAs are without excuse particularly in developed countries where there is never a reason to eat animal products.
Title: Re: Missing Link to Alzheimer's?
Post by: colporteur on January 08, 2017, 09:34:34 AM
     https://www.alz.org/braintour/plaques.asp


   Increasingly scientists are saying that Alzheimers is the result of a protein in the brain.

I vaguely recall a SOP quote where Mrs. White spoke of a terrible disease. What she described sounded  like it could be Alzheimer's. Of course there was no name attached to this disease back in her day.  Does anyone here remember that quote. It was kind of obscure.
Title: Re: Missing Link to Alzheimer's?
Post by: Richard Myers on January 08, 2017, 09:17:06 PM
A prion is an infectious agent composed entirely of protein material, called PrP (short for prion ... Prions may propagate by transmitting their misfolded protein state. I believe we have enough evidence to understand it is an infectious disease.
Title: Re: Missing Link to Alzheimer's?
Post by: colporteur on January 09, 2017, 05:50:00 AM

I read yesterday that Alzheimers has climbed to the 3rd leading cause if death in the US. While to jump to number 1 is a big jump considering how many people are dying from cancer and heart disease I cannot help but wonder if it is only a matter of time before that happens.
Title: Re: Missing Link to Alzheimer's?
Post by: Richard Myers on January 09, 2017, 08:02:57 AM
As prion diseases spread in animals, it will spread more rapidly to humans. But, then cancer will also. Few understand the connection between these diseases and the diseases in animals.
Title: Re: Missing Link to Alzheimer's?
Post by: rahab on March 06, 2020, 04:44:47 PM
It seems there have been medical reports going way back pointing to a cause, but it got pushed 'under the rug.' 

People that have clogged artery disease are prime victims for Alzheimer's disease.  Brain cannot get nutrients and Oxygen that it needs to function properly.  Some of this can be genetic and some just poor eating habits.
Title: Re: Missing Link to Alzheimer's?
Post by: Richard Myers on December 29, 2021, 09:11:27 PM
Amen!  With the understanding that amyloid is a protective agent it becomes easier to understand that many things can cause dementia including Alzheimer's. This video will help to better understand where science is headed in discovering the causes of this disease.






Here is an abstract revealing the amyloid found in sheep.

In prion and Alzheimer's diseases, the roles played by amyloid versus nonamyloid deposits in brain damage remain unresolved. In scrapie-infected transgenic mice expressing prion protein (PrP) lacking the glycosylphosphatidylinositol (GPI) membrane anchor, abnormal protease-resistant PrPres was deposited as amyloid plaques, rather than the usual nonamyloid form of PrPres. Although PrPres amyloid plaques induced brain damage reminiscent of Alzheimer's disease, clinical manifestations were minimal. In contrast, combined expression of anchorless and wild-type PrP produced accelerated clinical scrapie. Thus, the PrP GPI anchor may play a role in the pathogenesis of prion diseases.
Title: Re: Missing Link to Alzheimer's?
Post by: Richard Myers on December 29, 2021, 09:27:42 PM
Another discussion that is very interesting as it is explained that giving a drug would not fix a problem where many variables are involved. Very interesting that they came to the conclusion that lifestyle changes are necessary and can  bring reversal of dementia.