Author Topic: Mad Cows and People  (Read 39419 times)

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Richard Myers

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Re: Mad Cows and People
« Reply #100 on: January 16, 2011, 06:34:19 PM »
Researchers at the University of Zurich, the University Hospital Zurich and the University of Tübingen recommend precautionary measures for scientific labs, slaughterhouses and animal feed plants. New research suggest airborne prions are infectious and can induce mad cow disease or Creutzfeldt-Jakob Disease. Transmission of BSE to humans, e.g. by ingesting food derived from BSE-infected cows, causes variant Creutzfeldt-Jakob disease which is characterized by a progressive and invariably lethal break-down of brain cells.

It is known that prions can be transmitted through contaminated surgical instruments and, more rarely, through blood transfusions. The consumption of food products made from BSE-infected cows can also induce the disease that is responsible for the death of almost 300 people. However, prions are not generally considered to be airborne -- in contrast to many viruses including influenza and chicken pox.

Prof. Aguzzi says the findings are entirely unexpected and appear to contra-dict the widely held view that prions are not airborne. Not only are they infectious, but they are highly infectious.  source

The article did not mention the fact that mad cows and other material infected or thought to be infected have been incinerated. Generally when material is incinerated there is a release of material into the environment. Since they believed the prions to not be infectious when airborne, they may have misunderstood the risk from incineration.
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JimB

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Re: Mad Cows and People
« Reply #101 on: January 16, 2011, 07:06:31 PM »
Makes one wonder what the best way of disposal is so that it can't spread. Bury it?
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Mimi

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Re: Mad Cows and People
« Reply #102 on: January 16, 2011, 07:11:10 PM »
It should be treated as toxic waste.
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Richard Myers

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Re: Mad Cows and People
« Reply #103 on: January 16, 2011, 08:31:37 PM »
It is treated by most agencies in most countries as toxic waste. There are different levels of disposal for different agents. There are criteria for disposal, but they did not realize that the material was a danger in the air.  Apparently it is more toxic because there is no ability for the immune system to block it when breathed in as when eaten.

Prions are a very real problem. They are getting into the environment from cattle, sheep, deer, elk, cats, etc. They are in the soil and end up in water. I am not sure where it will end, but God knows. There is a topic on Chronic Wasting Disease (CWD) in this forum. It reveals the spread of this prion disease in elk and deer. It is spreading east and west on the northern US border.  We are living in the very last days of Earth's history.
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Richard Myers

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Re: Mad Cows and People
« Reply #104 on: April 02, 2011, 03:42:21 AM »
Some may feel very comfortable in eating animal products when they see no or little difficulty in their own country with vCJD. Let me exlain something that the world does not know, but that we do. vCJD has been directly tied in with the Mad Cows in the UK. But, there are other forms of Mad Cow Diseas in people besides vCJD. They are known as TSEs and are found in many animals. There is a CJD that is called "sporadic". It is not seen by most in government and industry to be communicated from animal to man so there is littel concern for preventing CJD unless it is vCJD.

That post was from 2003. This Australian report is dated June 2010.



Sporadic CJD, we were told "just happens". Note that this Australian doctor is saying that they now have evidence that sporadic CJD is being transmitted by Mad Cows to people. "The recent research is that in fact it is due to atypical forms of mad cow disease which have been found across Europe, have been found in America and have been found in Asia."  

Why is this important? Because the variant form of Mad Cows was what caused the CJD in England. We were told that the sCJD "just happens" so that it was not coming from mad sheep or mad cows. But, as I have been warning, the sporadic CJD (sCJD) is coming from eating animals and their products. The US has had very few cases of vCJD, but we have many cases of sCJD. Now the dots are being put together. Read through this thread and the CJD topic to get a better picture of the situation. It is much worse than we have been told.

And, this last week I had a doctor tell me that Alzheimer's is now being called a prion disease.  We have enough evidence to suggest that it also is a transmissible spongiform disease that is being introduced into humans via infected cows, deer, and sheep. The incubation period for these diseases can be very long or relatively short. See the Alzheimer's thread for more information.
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Mimi

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Re: Mad Cows and People
« Reply #105 on: April 02, 2011, 06:49:53 AM »
Quote
And, this last week I had a doctor tell me that Alzheimer's is now being called a prion disease.
Finally!
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Richard Myers

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Re: Mad Cows and People
« Reply #106 on: May 20, 2011, 04:13:14 PM »
For many who are not strict vegetarians, the scientific news is getting worse. They have now discovered that BSE (Mad Cow Disease) and Scrapie (Mad Sheep Disease) infects much more than the brain and nerve tissue.  A new study just published reveals that markers for the disease are found throughout the body.


Prions are largely contained within the nervous and lymphoid tissue of transmissible spongiform encephalopathy (TSE) infected animals. However, following advances in diagnostic sensitivity, PrPSc, a marker for prion disease, can now be located in a wide range of viscera and body fluids including muscle, saliva, blood, urine and milk, raising concerns that exposure to these materials could contribute to the spread of disease in humans and animals. Previously we demonstrated low levels of infectivity in the liver of sheep experimentally challenged with bovine spongiform encephalopathy. In this study we show that PrPSc accumulated in the liver of 89% of sheep naturally infected with scrapie and 100% of sheep challenged with BSE, at both clinical and preclinical stages of the disease. PrPSc was demonstrated in the absence of obvious inflammatory foci and was restricted to isolated resident cells, most likely Kupffer cells.
 
Citation: Everest SJ, Ramsay AM, Chaplin MJ, Everitt S, Stack MJ, et al. (2011) Detection and Localisation of PrPSc in the Liver of Sheep Infected with Scrapie and Bovine Spongiform Encephalopathy. PLoS ONE 6(5): e19737. doi:10.1371/journal.pone.0019737
 
Editor: Jason C. Bartz, Creighton University, United States of America
 
Received: December 30, 2010; Accepted: April 5, 2011; Published: May 12, 2011
 


The United States and other governments have insisted that the meat being sold is safe because it does not contain any "SRM", spcified risk material. This is meat that would contain nerve tissue. Japan refused to important any more US beef when they found a shipment that contained some specified risk material. Not all cows, sheep, goats, deer, or eld are infected with a spongiform disease, but the risk is steadily increasing. The evidence is pointing towards Alzheimer's being an infectious disease similar to CJD (human Mad Cow Disease) and animal Mad Cow and Scrapie Diseases.
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Richard Myers

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Re: Mad Cows and People
« Reply #107 on: February 01, 2012, 10:48:36 PM »
With BSE and other TSEs in animals, the recognition of the wide diversity of prion strains in the field, including three new forms of animal TSEs (L-type Atypical BSE, H-type Atypical BSE and Atypical scrapie), has complicated disease diagnosis and surveillance, as well as scientific assessment of their potential risks to humans. EFSA and the European Centre for Disease Prevention and Control (ECDC) recently delivered a scientific opinion on any possible epidemiological or molecular association between TSEs in animals and humans (EFSA Panel on Biological Hazards (BIOHAZ) and ECDC, 2011). This opinion confirmed Classical BSE prions as the only TSE agents demonstrated to be zoonotic so far but the possibility that a small proportion of human cases so far classified as “sporadic” CJD are of zoonotic origin could not be excluded. Moreover, transmission experiments to non-human primates suggest that some TSE agents in addition to Classical BSE prions in cattle (namely L-type Atypical BSE, Classical BSE in sheep, transmissible mink encephalopathy (TME) and chronic wasting disease (CWD) agents) might have zoonotic potential. In particular the L-type Atypical BSE agent might be similarly or even more virulent to humans than the Classical BSE agent. While mankind has been in contact with the major TSE of small ruminants for centuries, there is no epidemiological evidence to suggest that classical scrapie is zoonotic; however, experimental transmission data on humanised mice and non-human primates have been very scarce so far.  source
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Richard Myers

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Re: Mad Cows and People
« Reply #108 on: February 01, 2012, 10:55:02 PM »
While mankind has been in contact with the major TSE of small ruminants for centuries, there is no epidemiological evidence to suggest that classical scrapie is zoonotic; however, experimental transmission data on humanised mice and non-human primates have been very scarce so far. 

"No epidemiological evidence".  It is my understanding that lesions in sheep infected with scrapie are similar to amyloid plaques found in human dementia patients. The USDA has known this for over 20 years.
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Richard Myers

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Re: Mad Cows and People
« Reply #109 on: February 01, 2012, 11:31:29 PM »
I don't have time to spend doing research on this subject, but I took some time tonight ( it is late) to take a look at what has been happening.  I don't have to express my joy at finding this because those who have been following this thread already know.

Alzheimer's disease: A prion protein connection

Moustapha Cisse1 & Lennart Mucke1

February 26, 2009

More than 20 million people worldwide have Alzheimer's disease, yet its causes remain mostly uncertain. Fresh findings provide molecular clues, linking this disease to another neurodegenerative disorder.

Investigations of the causes of Alzheimer's disease yield one culprit time and time again: abnormal build-up of amyloid-β (Aβ) peptides in the brain. Small, soluble aggregates of Aβ — Aβ oligomers — impair memory by disrupting memory-related functions of synaptic junctions between neurons1, 2, 3.
  source

This finding came from the Gladstone Institute of Neurological Disease and in the Department of Neurology, University of California, San Francisco.  Alzheimer's disease: A prion protein connection means that there is substantial evidence pointing to Alzheimer's being caused by a spongiform disease.  Mad Cow, Scrapie, Chronic Wasting Disease, etc.
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Richard Myers

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Re: Mad Cows and People
« Reply #110 on: April 07, 2012, 03:04:34 PM »
Now that science is making headway into the dangers posed to humans by the spongiform diseases in animals, let's consider that while eating animal products is unsafe, there are other dangers also.  I have reported that research has shown that these diseases can be transmitted by bodily fluids including milk. And, it is believed that the infective agents can be found in soil also.

There is a study recently completed that will be interesting to those who are following this subject.

Currently, tests for CWD in live animals involve relatively invasive procedures to collect lymphoid tissue biopsies and examine them for Chronic Wasting Disease (Mad Elk and Deer).  Now, it has been shown that the "PMCA successfully detected PrPCWD in feces from elk that were positive by IHC, with estimated prion loads from 100 to 5,000 pg PrPCWD/g of feces. These data show for the first time PrPCWD in feces from naturally exposed free-ranging elk and demonstrate the potential of PMCA as a new, noninvasive CWD diagnostic tool to complement IHC." source

Detecting spongiform disease in live animals means that we ought to be able to detect spongiform disease in cows, sheep, and humans.  Prior to this research, it was an invasive procedure, but not any  longer. They found prion protein in feces. When this is available for sheep and cows, it could be disastrous for these two industries. Detecting BSE and Scrapie in live animals could reveal a much large infection rate than is currently estimated. Detection in humans could prevent spread of the disease through medical and dental procedures.

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Mimi

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Re: Mad Cows and People
« Reply #111 on: April 07, 2012, 03:40:00 PM »
The ramifications are huge. I see no end to it, do you?   

Here are other free-ranging animals reported as positive for CWD. http://www.cwd-info.org/
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Wally

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Re: Mad Cows and People
« Reply #112 on: April 07, 2012, 03:46:19 PM »
One of my long time customers was recently diagnosed with Creutzfeldt-Jakob disease.  I didn't realize that it was possible to diagnose it in a living person. They said that from the onset of symptoms until death is about 6 months.  The first symptoms appeared last fall, and he isn't expected to live much longer.  He is already unable to recognize anyone and is unable to function without full time care.  A very sad situation.  They don't know how he acquired the disease, but they are suggesting a hereditary factor.  I didn't have the heart to tell his wife that it could have come from eating infected meat.
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Mimi

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Re: Mad Cows and People
« Reply #113 on: April 07, 2012, 03:57:30 PM »
Very sad.

Soil clay content underlies prior infection. From what I know of soil composition in the Rocky Mountains, clay is king.

Environmental factors — especially soil properties — have been suggested as potentially important in the transmission of infectious prion diseases. Because binding to montmorillonite (an aluminosilicate clay mineral) or clay-enriched soils had been shown to enhance experimental prion transmissibility, we hypothesized that prion transmission among mule deer might also be enhanced in ranges with relatively high soil clay content. In this study, we report apparent influences of soil clay content on the odds of prion infection in free-ranging deer. Analysis of data from prion-infected deer herds in northern Colorado, USA, revealed that a 1 % increase in the clay-sized particle content in soils within the approximate home range of an individual deer increased its odds of infection by up to 8.9 % . Our findings suggest that soil clay content and related environmental properties deserve greater attention in assessing risks of prion disease outbreaks and prospects for their control in both natural and production settings.

http://www.wellsphere.com/cjd-article/soil-clay-content-underlies-prion-infection-odds/1369600
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Richard Myers

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Re: Mad Cows and People
« Reply #114 on: April 07, 2012, 04:01:55 PM »
It is very sad. Even more so when we realize the incubation period can be very long. People could have made changes ten years ago and still be infected from before then. Was it variant CJD or sporadic CJD? More than likely it is sporadic since they are saying it does not come from eating meat. It just happens. If it was variant, then more than likely he would have gotten it from the UK.  Although they have exported quite a bit of gelatin to the US that is a source of infection with variant CJD. If it is variant, it would have been widely reported.

They have convinced everyone that there is nothing that can be done about sporadic, so keep eating your animal products.
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Mimi

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Re: Mad Cows and People
« Reply #115 on: April 07, 2012, 04:22:28 PM »
Quote
Was it variant CJD or sporadic CJD?
I am not finding any of the reports from this site http://www.cwd-info.org/ that state which it is.

Am looking into the soil article, this one in particular: http://www.nature.com/ncomms/journal/v2/n2/pdf/ncomms1203.pdf
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Mimi

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Re: Mad Cows and People
« Reply #116 on: April 07, 2012, 04:34:41 PM »
Look at this short discussion on soil clay in the Rocky Mountains and the UK. More work needs to be done in that area:

Despite the plausibility, attempts to link specific soil characteristics
to patterns of prion disease occurrence at a broad geographical
scale have thus far yielded equivocal results 16,17 . Trace elements in
soils did not appear to explain observed patterns of scrapie occurrence
in the United Kingdom 17 , but soil clay mineralogy data were
not considered in that analysis. Analysis of various geochemical
factors and their respective influences on scrapie and bovine
spongiform encephalopathy incidences in Britain yielded relatively
weak evidence of soil clay and organic carbon contents increasing
or decreasing, respectively, disease incidence 16 . However, the
uniformly low-soil clay content ( < 5.2 % ) and low ratio of clay to
organic carbon ( < 2:1) in the soil data analysed, the small sample
sizes, and the coarse geographical scale 16 may have precluded or
obscured demonstrable effects. Moreover, the common practice of
adding phyllosilicate microparticles to livestock feeds 26 could overshadow
or confound the influences of soil-derived microparticles
on prion transmission risk in production setting as compared with
natural systems.


http://www.nature.com/ncomms/journal/v2/n2/pdf/ncomms1203.pdf
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Wally

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Re: Mad Cows and People
« Reply #117 on: April 08, 2012, 03:28:01 AM »
I don't know the type, but this man is originally from Denmark, so that may be a clue.
So likewise ye, when ye shall have done all those things which are commanded you, say, We are unprofitable servants:  we have done that which was our duty to do.  Luke 17:10

Wally

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Re: Mad Cows and People
« Reply #118 on: April 10, 2012, 05:13:44 PM »
I just got word yesterday that the man died.  It was inevitable, but very sad, nevertheless.  He and his wife had a very good relationship.  I can only hope that the Lord will be able use this tragedy as means of saving her.
So likewise ye, when ye shall have done all those things which are commanded you, say, We are unprofitable servants:  we have done that which was our duty to do.  Luke 17:10

Mimi

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Re: Mad Cows and People
« Reply #119 on: April 10, 2012, 06:20:56 PM »
It is sad. Much more education needs to be done in this area. People are needlessly dying.
  For ever, O LORD, thy word is settled in heaven. Psalm 119:89